“As a neuroprotective nutrient, lutein may support structure and function in neural membranes, and ultimately may support the cognitive functions that rely upon these neural membranes,” Marta Zamroziewicz, a graduate student in neuroscience and the study’s lead author, told The Huffington Post.
The link from the sun’s anti-entropic virucidal energy to lutein and cell type differentiation in all living genera was placed into the context of this review of human brain development.
The role of docosahexaenoic and the marine food web as determinants of evolution and hominid brain development: The challenge for human sustainability
The link from the sun’s anti-entropic virucidal energy to the de novo creation of olfactory receptor genes and to all biodiversity was placed into the context of this article: Feedback loops link odor and pheromone signaling with reproduction
“Evolutionary processes” fail to link docosahexanoic acid and lutein to luteinizing hormone and healthy longevity. The “processes” can be viewed in the context of what is known about energy-dependent biophysically constrained RNA-mediated protein folding chemistry. For example, energy is linked to healthy longevity in all living genera and virus-driven energy theft is linked to all pathology.
In all vertebrates, the stability of of organized genomes has been linked from the substitution of achiral glycine in position 6 of the luteinizing hormone releasing hormone (LHRH) decapeptide. The LHRH decapeptide is also referred to as the gonadotropin releasing hormone (GnRH) decapeptide.
The LHRH / GnRH decapeptide links dietary lutein to the nutrient-dependent pheromone-controlled cell type differentiation of all cell types in all vertebrates via the substitution of the achiral amino acid, glycine, and via measurements of luteinizing hormone, which initially was referred to as the “yellow hormone.”
See our section on “Tracking the Yellow Hormone” in: The Scent of Eros: Mysteries of Odor in Human Sexuality (1995/2002)
See also: From hydrogen atom transfer in DNA base pairs to ecosystems (video representation)
This atoms to ecosystems model of ecological adaptations links nutrient-dependent epigenetic effects on DNA base pairs in solution and RNA-mediated amino acid substitutions to chromosomal rearrangements via pheromone-controlled changes in the microRNA / messenger RNA balance. The nutrient-dependent pheromone-controlled changes are required for the thermodynamic regulation of intracellular signaling, which enables biophysically constrained nutrient-dependent & pH-dependent protein folding; experience-dependent receptor-mediated behaviors, and organism-level thermoregulation in ever-changing ecological niches and social niches. Critical limits for enhanced medical care already include what is known about the RNA-mediated physics and chemistry of biologically-based ecological, social, neurogenic and socio-cognitive niche construction. The epigenetic landscape is clearly linked to the physical landscape of supercoiled DNA and top-down causation is manifested in increasing organismal complexity in species from microbes to humans. In all vertebrates and invertebrates the reciprocal relationships of species-typical nutrient-dependent & pH-dependent morphological and behavioral diversity are enabled by microRNAs, adhesion proteins, and pheromone-controlled reproduction. Ecological variation and biophysically constrained natural selection of nutrients cause the RNA-mediated behaviors that enable ecological adaptations, which include development of the brain during life history transitions. Ideas from population genetics typically exclude ecological factors, which must be linked to cell type differentiation. Theories are integrated with an experimental evidence-based approach that establishes what is currently known in the context of this mammalian model.