A conserved NAD+ binding pocket that regulates protein-protein interactions during aging March 24, 2017 (Science)
Also reported as: It’s Happening: Scientists Can Now Reverse DNA Ageing in Mice
When we’re born, all of our cells have the ability to repair DNA damage, which we experience constantly through random mutations when our cells divide, or whenever we go out in the sun.
But as we get older, our ability to patch up this damage declines, and our cells being to age.
Before we get too excited, we need to keep in mind that many, many studies in mice are not replicated in humans.
So until the results of these early clinical trials in people begin to trickle in, there’s no promise that NMN will help protect human DNA.
DNA repair is energy-dependent and RNA-mediated in the context of the pheromone-controlled physiology of reproduction in all living genera. The physiology of reproduction links food odors and pheromones to the energy-dependent biophysically constrained RNA-mediated cell type differentiation.
See for comparison: Johns Hopkins researchers support mutation-driven evolution.