Epigenome-wide Association Studies and the Interpretation of Disease -Omics
Reported by Carl Zimmer in: Growing Pains for Field of Epigenetics as Some Call for Overhaul
Excerpt:
these experts “…argue that in some cases, changes to epigenetic marks don’t cause disease, but are merely consequences of disease.
My comment: What kind of biologically uninformed expert or science journalist continues to misrepresent what is known to serious scientists about all virus-driven pathology?
See for comparison: Epigenetics and Genetics of Viral Latency
Excerpt:
…viral latency is responsible for life-long pathogenesis and mortality risk…
See also: Mae-Wan Ho: No Boundary Really Between Genetic and Epigenetic
Excerpt:
…evolutionary science has now “moved on to such an extent” that she and Peter Saunders don’t really care anymore about “trying to convince the neo-Darwinists.”
Carl Zimmer and his “experts” continue to stand in the way of scientific progress. There is no such thing as an epigenome-wide association study outside the context of genome-wide association studies (GWAS) that link energy-dependent changes from angstroms to ecosystems in all living genera. The GWAS also link virus-driven energy theft to all pathology.
See: The phylogenetic utility and functional constraint of microRNA flanking sequences and RNA and dynamic nuclear organization
Energy-dependent RNA-mediated cell type differentiation is linked to all biodiversity via the innate immune system and supercoiled DNA. Virus-driven energy theft is linked from mutations to all pathology.