Yuste says he’s proud of what he and Denk accomplished. “But now I go to meetings where everyone is presenting two-photon calcium imaging, and no one knows, or remembers, or cites our work,” he says, laughing.
My comment: What else can be done but laugh? Most researchers still have not linked the anti-entropic energy of virucidal ultraviolet light from the biophysically constrained RNA-mediated chemistry of protein folding to all biodiversity in all genera despited the advances that link biophotonics and femotosecond blasts of UV light to DNA repair and to supercoiled DNA via the physiology of reproduction.
Many still seem to be largely unaware that supercoiled DNA protects all organized genomes from virus-driven entropy because they cannot see the femtosecond blasts. They decide to link mutations to the evolution of all biodiversity, instead.
See for comparison the molecular epigenetics section of our 1996 Hormones and Behavior review. From Fertilization to Adult Sexual Behavior
I stopped attending meetings where everyone is presenting on molecular epigenetics and no one knows, or remembers, or cites our work. Neo-Darwinian theorists have continued to present findings in the context of pseudoscientific nonsense and the are now trying to link the molecular mechanisms of energy-dependent polycombic ecological adaptation to a new ridiculous theory of evolution, which they refer to as adaptation.
The unifying theme for much of modern biology is based on Charles Darwin’s theory of evolution, the process of natural selection by which nature selects the fittest, best-adapted organisms to reproduce, multiply and survive. The process is also called adaptation, and traits most likely to help an individual survive are considered adaptive.
My comment: The process of evolution was not called adaptation until people like Michael Skinner realized there is no such thing as mutation-driven evolution. They know it is obvious that nutrient energy-dependent pheromone-controlled biophysically constrained protein folding chemistry controls cell type differentiation because otherwise life could not exist on Earth.
See also: Why do some cancers suddenly disappear?
Glutamine is the amino acid coded for by the genomic trinucleotide CAG. Repeating glutamines, called polyglutamines, are normal in huntingtin proteins, but when the DNA is copied incorrectly, the repeating sequence of glutamines can become too long. The result can be diseases like Huntington’s or spinocerebellar ataxia.
The number of repeats of glutamine can grow as the genetic code information is passed down through generations. That means a healthy parent whose huntingtin gene encodes proteins with 35 repeats may produce a child with 36 repeats. A person having the longer repeat is likely to develop Huntington’s disease.
My comment: The number of repeats is nutrient energy-dependent and longer repeats link virus-driven energy theft to all pathology.