How many proponents of Intelligent Design never learned that Darwinian evolution was based on his “conditions of life?”
ch 4 IV. Natural Selection; or the Survival of the Fittest “IF under changing conditions of life…”
ch 5 http://www.bartleby.com/11/5002.html “…the greater variability of species having wider ranges than of those with restricted ranges, lead to the conclusion that variability is generally related to the conditions of life…”
“…we cannot tell how much to attribute to the accumulative action of natural selection, and how much to the definite action of the conditions of life.”
ch 6 http://www.bartleby.com/11/6001.html “By my theory these allied species are descended from a common parent; and during the process of modification, each has become adapted to the conditions of life…
How many biologically uninformed science idiots do not know that conditions of life are nutrient energy-dependent and controlled by the physiology of reproduction?
Food energy-dependent RNA-directed DNA methylation alters the shifting of membrane helices. The alterations prevent the virus-driven degradation of messenger RNA and all pathology via fixation of amino acid substitutions in organized genomes. The physiology of reproduction and pheromone-controlled chromosomal inheritance link the fixation of amino acid substitutions in all cell types of all living genera to all biodiversity.
Yoshinori Ohsumi won last year for doing that: A protein conjugation system essential for autophagy
This [energy-dependent] conjugation can be reconstituted in vitro and depends on ATP.
Ben Feringa also won last year: Dynamic control of chirality and self-assembly of double-stranded helicates with light
All serious scientists are waiting for all pseudoscientists to realize the level of ignorance they have displayed for at least the past 50 years and take responsibility for the unnecessary suffering and premature deaths their ridiculous theories have caused.
Thank God, some serious scientists have taken the lead.
Pathway analyses found a significant enrichment for “xenobiotic metabolism signaling” and “PXR/RXR activation” pathways. Our H3K27ac ChIP-seq identified 9,514 peaks that are PenStrep responsive. These peaks were enriched near genes that function in cell differentiation, tRNA modification, nuclease activity and protein dephosphorylation. Our results suggest that PenStrep treatment can significantly alter gene expression and regulation in a common liver cell type such as HepG2, advocating that antibiotic treatment should be taken into account when carrying out genetic, genomic or other biological assays in cultured cells.
Admitting that you and your colleagues once were biologically uninformed science idiots is the first step towards recovery. The antibiotic treatment caused the constraint-breaking mutations that all serious scientists have linked to all pathology. Food energy and the pheromone-controlled physiology of reproduction link biophysically constrained viral latency to healthy longevity.