Nutrient-dependent life vs virus-driven pathology

James, you are one step away from being banned because of your disingenuous comments, your accusations, and your unnecessary combativeness. I am getting tired of it. People accuse you of being a troll for a reason.
This is your final warning.
My comment: Thanks for the warning, Perry Marshall. People who do not like me to disseminate accurate information about biologically-based cause and effect accuse me of being a troll. Is that what you are doing?
See also: How Answers in Genesis turns Home Schoolers into atheists

Perry Marshall continues to tout pseudoscientific nonsense and criticize others who don’t accept his ridiculous claims.
See, for comparison:. Identification of Amino Acid Substitutions Supporting Antigenic Change of Influenza A(H1N1)pdm09 Viruses

The high mutation rate of influenza A viruses and the observation that single substitutions caused substantial antigenic change during evolution of A(H3N2), A(H1N1), and influenza B viruses contradict the relatively low rate at which influenza viruses have changed antigenically (11).

My comment: Koel et al., (2013)  admitted that  The major antigenic changes of the influenza virus are primarily caused by a single amino acid near the receptor binding site. Earlier today, I decided to see if BF Koel, or any of his co-authors had learned how nutrient-dependent cell type differentiation links atoms to ecosystems.
Like Perry Marshall, they still appear to be horribly confused. They seem to think that a mutation is the same thing as a nutrient-dependent RNA-mediated amino acid substitution.  I suspect that their confusion is the common theme that prevents scientific progress from eliminating neo-Darwinian nonsense about beneficial mutations and evolution.
See for comparison: Friends with (Brain) Benefits: The impact of the gut microbiota on neurodevelopment (The presentation is free and available on demand until September, 2016)
My comment:  John Cryan explained more about biologically-based cause and effect than any of the presenters whose works should also be examined in the light of his claims. Finally, I’ve seen evidence presented that clearly links metabolic networks to genetic networks via nutrient-dependent microRNAs, adhesion proteins, and the physiology of reproduction, which must link energy-dependent changes in base pairs from RNA-mediated amino acid substitutions to supercoiled DNA.
See also: Human milk miRNAs primarily originate from the mammary gland resulting in unique miRNA profiles of fractionated milk

Given the recently identified stability, uptake and functionality of food- and milk-derived miRNA in vivo, HM miRNA are likely to contribute to infant protection and development.

My comment: The microRNAs in human milk establish the connections from the microRNAome of bull sperm and the physiology of reproduction in soil bacteria to UV-light induced RNA-mediated nutrient-dependent DNA repair in plants and animals.
The neuroscientifically established connections link supercoiled DNA to protection from virus-driven entropy. It may be interesting for others to learn how many established facts Dr. Cryan can present within established time constraints.
See also: Milk: an epigenetic amplifier of FTO-mediated transcription? Implications for Western diseases

Milk is a highly specialized nutrient and signaling system of mammalian evolution that apparently shapes the epitranscriptome of the milk recipient via FTO-mediated modifications of RNA nucleotides. Future research should unravel milk´s biological impact on the recently recognized dual axis of coordinated regulation between the genome and the epigenome and the transcriptome and the epitranscriptome, respectively [336].

See also:
Friends with social benefits: host-microbe interactions as a driver of brain evolution and development?
Microbial genes, brain & behaviour – epigenetic regulation of the gut–brain axis
The brain’s Geppetto—microbes as puppeteers of neural function and behaviour?
Reported as: The master puppeteer? – Impact of microbes on brain and behaviour
See also:
Early-life stress-induced visceral hypersensitivity and anxiety behavior is reversed by histone deacetylase inhibition
Downregulation of Umbilical Cord Blood Levels of miR-374a in Neonatal Hypoxic Ischemic Encephalopathy
Adolescent behavior and dopamine availability are uniquely sensitive to dietary omega-3 fatty acid deficiency
Gestational Stress Induces Depressive-like and Anxiety-like Phenotypes through Epigenetic Regulation of BDNF Expression in Offspring Hippocampus
The maternal microbiota drives early postnatal innate immune development
Reported 3/17/16 as: What’s in the breast milk affects microbes in the baby
My comment: The maternal microRNAome and paternal microRNAome establish the innate immune system and metabolic networks linked to genetic networks of human infants via nutrient-dependent RNA-mediated DNA repair in the context of the physiology of reproduction linked to supercoiled DNA and transgenerational epigenetic inheritance of morphological and behavioral phentotypes in all invertebrates and vertebrates. If not for neo-Darwinian pseudoscience, the link from virus-driven energy theft would have been examined in the context of all pathology, and most pathology might already have been eliminated. All pathology could be more effectively treated if only the serious scientists who know how cell type differentiation occurs could have prevailed against the ignorance of theorists who think that beneficial mutations can lead to evolution of one species from another — given enough time.
Presented 3/16/16 and 3/17/16 See the Poster Hall at the Labroots Neuroscience 2016 Virtual Event
In the Neuron Biology section, see:

1) From hydrogen-atom transfer in DNA base pairs to ecosystems
2) The Origin of Information [RNA-mediated molecular epigenetics and virus-driven entropy]
For additional information presented on 3/16 and 3/17 that indirectly links hydrogen- atom transfer in DNA base pair in solution to supercoiled DNA, which protects all organized genomes from virus-driven entropy, see the presentations by: Geraint Wilde, Fred H Gage, and Kathleen Gough at the Labroots Neuroscience 2016 Virtual Event.

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