For review see my blog posts “The tipping point? 50,000 publications” with follow-ups: (2) (3) & (4).
There are now more than 52,000 published works that link microRNAs to cell type differentiation in the context of healthy longevity or virus-driven energy theft, which has been linked from viral latency to all pathology.
At the rate of ~1000 more publications per month that link energy-dependent changes in the microRNA/messenger RNA balance to healthy longevity or link virus-driven energy theft to all pathology, it won’t be much longer until public outcry puts an end to the money-grabbing evolution industry and big bang cosmology industry support of theories.
Combating Evolution to Fight Disease is the next step forward to altering the progress of pathology. Others have learned enough about RNA-mediated cell type differentiation to move science forward by leaving neo-Darwinian pseudoscientific nonsense behind.
See: Life’s Greatest Secret: The Race to Crack the Genetic Code
“Information” was about to enter science, but had not done so when Schrödinger gave his lectures. Without that conception of the content of the code, Schrödinger’s insight was merely part of the zeitgeist, a hint of what was to come rather than a breakthrough that shaped all subsequent thinking.– Matthew Cobb (2015)
See for comparison:
I am absolutely certain that if you showed this statement to any professor of biology or genetics in any accredited university anywhere in the world that 100% of them would say that “Random mutations are the substrate upon which directional natural selection acts” is a correct and true statement. — Jay R. Feierman (2013)
My comment: Schrodinger’s published claims (1944) set the stage for discussions about energy as information in the context of reality. His equation helped those who were interested in discussion. His claim about the link from sunlight as the source of fuel for all biodiversity is rarely mentioned. Feierman, for example, seems to think that random mutations can somehow be linked from natural selection to all biodiversity.
See for comparison: “What is Life?“
Indeed, in the case of higher animals we know the kind of orderliness they feed upon well enough, viz. the extremely well-ordered state of matter in more or less complicated organic compounds, which serve them as foodstuffs. After utilizing it they return it in a very much degraded form -not entirely degraded, however, for plants can still make use of it. (These, of course, have their most power supply of ‘negative entropy’ the sunlight)
See also the forward by Roger Penrose
How often do we still hear that quantum effects can have little relevance in the study of biology, or even that we eat food in order to gain energy?
My comment: If you can link the anti-entropic virucidal energy of the sun to hydrogen-atom transfer in DNA base pairs in solution, you can link ultraviolet light to RNA-mediated DNA repair via the innate immune system, the physiology of reproduction, and supercoiled DNA, which typically protects us from all virus-driven energy theft and pathology.
The energy as information is linked from RNA methylation to learning and memory at the cellular and organismal level is the basis for this patent application: RNA-Guided Human Genome Engineering
Repetitive elements or endogenous viral elements can be targeted with engineered Cas+gRNA systems in microbes, plants, animals, or human cells to reduce deleterious transposition or to aid in sequencing or other analytic genomic/transcriptomic/proteomic/diagnostic tools (in which nearly identical copies can be problematic).
My comment: Latent viruses (i.e., endogenous viral elements) are the obvious source of all pathology. See: “…viral latency is responsible for life-long pathogenesis and mortality risk…” Paul M. Lieberman
If a patent is issued for RNA-Guided Human Genome Engineering, the technology may never be available to all those who need it.
Indeed, the people who need the technology most may never be told why they need it. They will need it because the nutrient-dependent pheromone-controlled physiology of energy-dependent reproduction has been altered by the use of viruses to genetically modify the nutritional value of plants that people eat for food. Now others have learned that virus altered nutrient stress and social stress make the largest contribution to how and why viral latency becomes life-long pathogenesis and mortality risk. Apparently, they intend to make billions with technology that uses RNA to repair DNA, which is what our innate immune system always has done. The RNA-mediated repair is their RNA-Guided Human Genome Engineering.
See also: N6-methyladenosine of HIV-1 RNA regulates viral infection and HIV-1 Gag protein expression
These findings suggest that adenosine methylation plays an important role in regulating the ability of HIV to thrive and multiply in T-lymphocytes, which are an important target for HIV. Since the RNAs of other human viruses, such as influenza virus, can also be modified by adenosine methylation, drugs that target this pathway could have the potential to be used to fight a variety of viral illnesses.
My comment: The fact that they have linked energy-dependent RNA methylation to RNA-directed DNA methylation and healthy longevity via prevention of virus-driven energy theft is presented in the context of drug development, which is typical. No one makes much money by reporting how energy-dependent cell type differentiation is linked to healthy longevity. For comparison, the RNA-Guided Human Genome Engineering patent may be worth billions.
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