Virus-driven cancer treatment and prevention

Blocking a metabolic pathway may shrink aggressive form of common kidney cancer


..the cancer cells appear to rely primarily on the metabolism of an amino acid called glutamine for their energy.

My comment:  This links differences in hydrogen atom energies and the anti-entropic epigenetic effect of nutrient energy to virus-driven differences in the metabolism of glutamine and DNA repair. Those differences link viral microRNAs to entropic elasticity. The differences also link nutrient-dependent microRNAs to the stability of organized genomes via RNA-directed DNA methylation and RNA-mediated amino acid substitutions.
Thermodynamic cycles of protein biosynthesis and degradation that prevent genetic entropy are perturbed by viral microRNAs. That is why models of cancer must start with links from atoms to ecosystems that link what is currently known to serious scientists about the conserved molecular mechanisms of biophysically constrained RNA-mediated amino acid substitutions and protein folding during the life history transitions of all genera.
Simply put, researchers must learn how cell type differentiation occurs before they can begin to understand what goes wrong. What goes wrong links viral microRNAs from perturbed protein folding to pathology via RNA-mediated events that link metabolic networks to genetic networks.

Researchers identify cell‑changing gene that can cause cancer


Herpesvirus-8 targets cells that line the surface of blood and lymphatic vessels. Using one of its genes, Kaposin B, the virus reprograms these cells. It gives the cells instructions to grow, form new vessels, and inflame surrounding tissues. These changes in the cells can lead to cancer.

My comment: You probably got the impression that a gene caused cancer. That is what evolutionary theorists would like you to think.  If you continue to believe in their pseudoscientific nonsense, the evolution industry and the big bang cosmology industry will triumph over common sense. For example:  “…genomic conservation and constraint-breaking mutation is the ultimate source of all biological innovations and the enormous amount of biodiversity in this world. In this view of evolution there is no need of considering teleological elements” (p. 199).
For comparison: “… the massive creative power of a cooperative RNA consortium (QS-C) remains crucial for life. QS-C was made known to us only recently by virus evolution (e.g., HIV-1). Its role in the origin of life, the emergence of complexity and the creation of group identity should now receive our combined attention” (p. 8).
See also:

Nutrient-dependent pheromone-controlled ecological adaptations: from atoms to ecosystems

Abstract: This atoms to ecosystems model of ecological adaptations links nutrient-dependent epigenetic effects on base pairs and amino acid substitutions to pheromone-controlled changes in the microRNA / messenger RNA balance and chromosomal rearrangements. The nutrient-dependent pheromone-controlled changes are required for the thermodynamic regulation of intracellular signaling, which enables biophysically constrained nutrient-dependent protein folding; experience-dependent receptor-mediated behaviors, and organism-level thermoregulation in ever-changing ecological niches and social niches. Nutrient-dependent pheromone-controlled ecological, social, neurogenic and socio-cognitive niche construction are manifested in increasing organismal complexity in species from microbes to man. Species diversity is a biologically-based nutrient-dependent morphological fact and species-specific pheromones control the physiology of reproduction. The reciprocal relationships of species-typical nutrient-dependent morphological and behavioral diversity are enabled by pheromone-controlled reproduction. Ecological variations and biophysically constrained natural selection of nutrients cause the behaviors that enable ecological adaptations. Species diversity is ecologically validated proof-of-concept. Ideas from population genetics, which exclude ecological factors, are integrated with an experimental evidence-based approach that establishes what is currently known. This is known: Olfactory/pheromonal input links food odors and social odors from the epigenetic landscape to the physical landscape of DNA in the organized genomes of species from microbes to man during their development.

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