We house our animal mouse models in pathogen free environments (aside from transponson). They still age and die with pathological features. Sorry
On Saturday, 4/22/17, which was the day of the “March for Science” he wrote:
You should not have featured me. It is out of context and you have no idea what you’re talking about. You just want there to be a god like the other children . Leave me out of this quackery. Remove my name. Never contact me.
Excerpt: There’s an old legal proverb:
If you have the facts on your side, argue the facts. If you have the law on your side, argue the law. If you have neither, attack the witness. When proponents of a scientific consensus lead with an attack on the witness, rather than on the arguments and evidence, be suspicious.
Chad Lerner attacked me as if I was a witness for Creationism and Americanism. Tomorrow is another day.
See Behavior related genes, dietary preferences and anthropometric traits,which is scheduled for presentation on 4/24/17
Reported as: Could genetics influence what we like to eat?
…little is known about how natural variation in these genes could affect eating behaviors in healthy people. Gene variation is a result of subtle DNA differences among individuals that make each person unique.
All serious scientists know that natural variation in genes is nutrient energy-dependent and controlled by the biophysically constrained physiology of reproduction. The also know this: Feedback loops link odor and pheromone signaling with reproduction
Natural selection for energy-dependent codon optimality links the feedback loops to all biophysically constrained biologically-based cause and effect. There is a model for that: Nutrient-dependent/pheromone-controlled adaptive evolution: a model.
This claim makes no sense: “…little is known about how natural variation in these genes could affect eating behaviors in healthy people.”